Charlotte Schubert is a freelance science journalist based in Seattle with previous staff experience at Nature Medicine and at the Milwaukee Journal Sentinel. She has written for Nature, Proceedings of the National Academy of Sciences, Cell Press and other publications. She has Ph.D. from the University of Washington and just completed a year-long stint in an epigenetics lab at Fred Hutchinson Cancer Research Center.
Charlotte Schubert
From this contributor
Genetic risk factors for autism may affect family size
People who carry risk factors for autism but do not have the condition tend to have slightly fewer children than average, and have them later in life.
Genetic risk factors for autism may affect family size
Explore more from The Transmitter
Infant Brain Imaging Study findings, and more
Here is a roundup of autism-related news and research spotted around the web for the week of 23 March.
Infant Brain Imaging Study findings, and more
Here is a roundup of autism-related news and research spotted around the web for the week of 23 March.
Trading places: What happens when neuroscience turns into machine learning, and machine learning turns into neuroscience?
Neuroscience has become increasingly concerned with prediction, and machine learning with causal explanation, with each field adopting methods from the other. I asked eight experts to weigh in on what we stand to learn from this exchange.
Trading places: What happens when neuroscience turns into machine learning, and machine learning turns into neuroscience?
Neuroscience has become increasingly concerned with prediction, and machine learning with causal explanation, with each field adopting methods from the other. I asked eight experts to weigh in on what we stand to learn from this exchange.
Exon-skipping approach boosts levels of key Rett syndrome protein
Deleting a small region of the MECP2 gene partially restored function in neurons derived from people with Rett-associated variants.
Exon-skipping approach boosts levels of key Rett syndrome protein
Deleting a small region of the MECP2 gene partially restored function in neurons derived from people with Rett-associated variants.