I am a postdoctoral fellow in the lab of Arnold Kriegstein at the University of California, San Francisco (UCSF). I focus on applying single-cell genomics techniques to study the development of specific cell types of the human brain, as well as to understand how these cell types are affected in various diseases, especially autism. Before starting my work at UCSF, I did my PhD at the University of Miami focusing on genomic analysis of autism. I did my B.S. and MS at Moscow State University in my native Russia, where I worked on animal models of epilepsy and Alzheimer’s disease.
Dmitry Velmeshev
Postdoctoral Scholar
University of California, San Francisco
From this contributor
Single-cell analysis suggests brain signaling problems in autism
Recent advances in technology allow researchers to measure RNA that is contained within the nucleus of a single brain cell.

Single-cell analysis suggests brain signaling problems in autism
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Food for thought: Neuronal fuel source more flexible than previously recognized
The cells primarily rely on glucose—rather than lactate from astrocytes—to generate energy, according to recent findings in mice.

Food for thought: Neuronal fuel source more flexible than previously recognized
The cells primarily rely on glucose—rather than lactate from astrocytes—to generate energy, according to recent findings in mice.
Claims of necessity and sufficiency are not well suited for the study of complex systems
The earliest studies on necessary and sufficient neural populations were performed on simple invertebrate circuits. Does this logic still serve us as we tackle more sophisticated outputs?

Claims of necessity and sufficiency are not well suited for the study of complex systems
The earliest studies on necessary and sufficient neural populations were performed on simple invertebrate circuits. Does this logic still serve us as we tackle more sophisticated outputs?
Subthalamic plasticity helps mice squelch innate fear responses
When the animals learn that a perceived threat is not dangerous, long-term activity changes in a part of the subthalamus suppress their instinctive fears.

Subthalamic plasticity helps mice squelch innate fear responses
When the animals learn that a perceived threat is not dangerous, long-term activity changes in a part of the subthalamus suppress their instinctive fears.