Mustafa Sahin is professor of neurology at Harvard University and director of the Translational Neuroscience Center at Boston Children’s Hospital.
Mustafa Sahin
Professor
Harvard University
From this contributor
Studies of tuberous sclerosis may shed light on biology of autism
Tuberous sclerosis provides a unique opportunity to understand autism because about half of people with that single-gene condition also have autism.
Studies of tuberous sclerosis may shed light on biology of autism
By
Mustafa Sahin
24 September 2019 | 5 min read
Insights for autism from tuberous sclerosis complex
Studying tuberous sclerosis provides researchers with a unique opportunity to find a common pathway among the various genetic causes of autism, says neurologist Mustafa Sahin.
Insights for autism from tuberous sclerosis complex
By
Mustafa Sahin
24 July 2012 | 6 min read
Explore more from The Transmitter
Human brain organoids flourish in emptied mouse cortex
The chimeric mice could help untangle the biology of human brain development or conditions such as autism.
Human brain organoids flourish in emptied mouse cortex
The chimeric mice could help untangle the biology of human brain development or conditions such as autism.
By
Brianna Abbott
16 September 2026 | 6 min read
What if Hegel were a neuroscientist?
Andrea Gambarotto shares what Hegel and enactivism have in common: an agency-first approach to understanding minds and brains.
What if Hegel were a neuroscientist?
Andrea Gambarotto shares what Hegel and enactivism have in common: an agency-first approach to understanding minds and brains.
By
Paul Middlebrooks
16 September 2026 | 1 min read
This paper changed my life: Transformative work in addiction neuroscience
A 2011 Nature study found that loss of a nicotinic receptor subunit leads to increased nicotine intake in mice. The work set the standard for how to functionally validate genetic association study findings in neuroscience.
This paper changed my life: Transformative work in addiction neuroscience
A 2011 Nature study found that loss of a nicotinic receptor subunit leads to increased nicotine intake in mice. The work set the standard for how to functionally validate genetic association study findings in neuroscience.
By
Jibran Khokhar
16 September 2026 | 6 min read