James Harris is professor of psychiatry and behavioral sciences at Johns Hopkins University School of Medicine in Baltimore, where he is founding director of the Developmental Neuropsychiatry Clinic. He is also a professor at Johns Hopkins Bloomberg School of Public Health. He served on the American Psychiatric Association committee that wrote the new definition of autism spectrum disorder for the DSM-5.
James Harris
Professor of psychiatry and behavioral sciences
Johns Hopkins University School of Medicine
From this contributor
Correcting the record: Leo Kanner and the broad autism phenotype
The specter of the ‘refrigerator mother’ theory continues to haunt the history of autism. New information puts Kanner’s observations of parents into context.
Correcting the record: Leo Kanner and the broad autism phenotype
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Dose, scan, repeat: Tracking the neurological effects of oral contraceptives
We know little about how the brain responds to oral contraceptives, despite their widespread use. I am committed to changing that: I scanned my brain 75 times over the course of a year and plan to make my data openly available.
Dose, scan, repeat: Tracking the neurological effects of oral contraceptives
We know little about how the brain responds to oral contraceptives, despite their widespread use. I am committed to changing that: I scanned my brain 75 times over the course of a year and plan to make my data openly available.
Cracking the code of the extracellular matrix
Despite evidence for a role in plasticity and other crucial functions, many neuroscientists still view these proteins as “brain goop.” The field needs technical advances and a shift in scientific thinking to move beyond this outdated perspective.
Cracking the code of the extracellular matrix
Despite evidence for a role in plasticity and other crucial functions, many neuroscientists still view these proteins as “brain goop.” The field needs technical advances and a shift in scientific thinking to move beyond this outdated perspective.
Huntington’s disease gene variants past a certain size poison select cells
The findings—providing “the next step in the whole pathway”—help explain the disease’s late onset and offer hope that it has an extended therapeutic window.
Huntington’s disease gene variants past a certain size poison select cells
The findings—providing “the next step in the whole pathway”—help explain the disease’s late onset and offer hope that it has an extended therapeutic window.