Rebecca Saxe’s work addresses the human brain’s capacity for abstract thought and the origins of ‘theory of mind,’ the ability to understand the beliefs, hopes and plans of other people.
Rebecca Saxe
Professor
Massachussetts Institute of Technology
From this contributor
U.S. agency backtracks on broad interpretation of ‘clinical trial’
Autism researchers need no longer worry that their basic research will become entangled in the red tape associated with clinical trials.
U.S. agency backtracks on broad interpretation of ‘clinical trial’
1985 paper on the theory of mind
In 1985, Simon Baron-Cohen, Alan Leslie and Uta Frith reported for the first time that children with autism systematically fail the false belief task.
Explore more from The Transmitter
Dose, scan, repeat: Tracking the neurological effects of oral contraceptives
We know little about how the brain responds to oral contraceptives, despite their widespread use. I am committed to changing that: I scanned my brain 75 times over the course of a year and plan to make my data openly available.
Dose, scan, repeat: Tracking the neurological effects of oral contraceptives
We know little about how the brain responds to oral contraceptives, despite their widespread use. I am committed to changing that: I scanned my brain 75 times over the course of a year and plan to make my data openly available.
Cracking the code of the extracellular matrix
Despite evidence for a role in plasticity and other crucial functions, many neuroscientists still view these proteins as “brain goop.” The field needs technical advances and a shift in scientific thinking to move beyond this outdated perspective.
Cracking the code of the extracellular matrix
Despite evidence for a role in plasticity and other crucial functions, many neuroscientists still view these proteins as “brain goop.” The field needs technical advances and a shift in scientific thinking to move beyond this outdated perspective.
Huntington’s disease gene variants past a certain size poison select cells
The findings—providing “the next step in the whole pathway”—help explain the disease’s late onset and offer hope that it has an extended therapeutic window.
Huntington’s disease gene variants past a certain size poison select cells
The findings—providing “the next step in the whole pathway”—help explain the disease’s late onset and offer hope that it has an extended therapeutic window.