Richard Bethlehem is a postdoctoral fellow and research associate at the Autism Research Centre and Brain Mapping Unit at the University of Cambridge in the United Kingdom. He studies integrated neuroimaging and transcriptomics to gain better understanding of the biological underpinnings of typical and atypical neurodevelopment.
Richard Bethlehem
Research associate
University of Cambridge
From this contributor
Q&A with Richard Bethlehem: What goes into a Brainhack
Brainhack conferences offer talks and hands-on tutorials, and unite small groups of interdisciplinary researchers to work on open-source neuroscience projects.
Q&A with Richard Bethlehem: What goes into a Brainhack
How normative modeling can reframe autism’s heterogeneity
Normative modeling could capture variability among autistic people and allow for individualized assessments.
How normative modeling can reframe autism’s heterogeneity
Explore more from The Transmitter
Infant Brain Imaging Study findings, and more
Here is a roundup of autism-related news and research spotted around the web for the week of 23 March.
Infant Brain Imaging Study findings, and more
Here is a roundup of autism-related news and research spotted around the web for the week of 23 March.
Trading places: What happens when neuroscience turns into machine learning, and machine learning turns into neuroscience?
Neuroscience has become increasingly concerned with prediction, and machine learning with causal explanation, with each field adopting methods from the other. I asked eight experts to weigh in on what we stand to learn from this exchange.
Trading places: What happens when neuroscience turns into machine learning, and machine learning turns into neuroscience?
Neuroscience has become increasingly concerned with prediction, and machine learning with causal explanation, with each field adopting methods from the other. I asked eight experts to weigh in on what we stand to learn from this exchange.
Exon-skipping approach boosts levels of key Rett syndrome protein
Deleting a small region of the MECP2 gene partially restored function in neurons derived from people with Rett-associated variants.
Exon-skipping approach boosts levels of key Rett syndrome protein
Deleting a small region of the MECP2 gene partially restored function in neurons derived from people with Rett-associated variants.