Stormy Chamberlain is associate professor of genetics and genome sciences and associate director of the Graduate Program in Genetics and Developmental Biology at the University of Connecticut School of Medicine.

Stormy Chamberlain
Associate professor
University of Connecticut
From this contributor
Angelman syndrome’s silent gene points way forward for autism therapies
Advances in research and help from families have brought scientists to the brink of an effective therapy for Angelman syndrome.

Angelman syndrome’s silent gene points way forward for autism therapies
For accurate results in autism, genetic databases need diversity
We must diversify databases of reference DNA to improve our ability to interpret the consequences of genetic variation.

For accurate results in autism, genetic databases need diversity
Explore more from The Transmitter
Bespoke photometry system captures variety of dopamine signals in mice
The tool tracks the excitation of an engineered protein that senses dopamine’s absolute levels, including fast and slow fluctuations in real time, and offers new insights into how the signals change across the brain.

Bespoke photometry system captures variety of dopamine signals in mice
The tool tracks the excitation of an engineered protein that senses dopamine’s absolute levels, including fast and slow fluctuations in real time, and offers new insights into how the signals change across the brain.
What infant fMRI is revealing about the developing mind
Cognitive neuroscientists have finally clocked how to perform task-based functional MRI experiments in awake babies—long known for their inability to lie still or take direction. Next, they aim to watch cognition take shape and settle a debate about our earliest memories—with one group publishing a big clue today.

What infant fMRI is revealing about the developing mind
Cognitive neuroscientists have finally clocked how to perform task-based functional MRI experiments in awake babies—long known for their inability to lie still or take direction. Next, they aim to watch cognition take shape and settle a debate about our earliest memories—with one group publishing a big clue today.
Molecular changes after MECP2 loss may drive Rett syndrome traits
Knocking out the gene in adult mice triggered up- and down-regulated expression of myriad genes weeks before there were changes in neuronal function.

Molecular changes after MECP2 loss may drive Rett syndrome traits
Knocking out the gene in adult mice triggered up- and down-regulated expression of myriad genes weeks before there were changes in neuronal function.