Christian Schaaf is professor of human genetics at the University of Heidelberg in Germany.

Christian Schaaf
Professor
University of Heidelberg
From this contributor
Oxytocin lessons from autism-linked syndromes: A chat with Christian Schaaf and Ferdinand Althammer
Oxytocin therapies have failed to consistently benefit autistic people, but their effects in people with two autism-linked conditions may yield new insights, experts argue.

Oxytocin lessons from autism-linked syndromes: A chat with Christian Schaaf and Ferdinand Althammer
How an expert panel evaluates genes for autism genetic tests
About 15 percent of genes currently included in clinical genetic tests for autism or intellectual disability don’t have enough evidence to support their ties to the conditions, the panel found.

How an expert panel evaluates genes for autism genetic tests
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Fly database secures funding for another year, but future remains in flux
The FlyBase team’s fundraising efforts have proven successful in the short term, but restoration of its federal grant remains uncertain.

Fly database secures funding for another year, but future remains in flux
The FlyBase team’s fundraising efforts have proven successful in the short term, but restoration of its federal grant remains uncertain.
Diving in with Nachum Ulanovsky
With an eye toward realism, the neuroscientist, who has a new study about bats out today, creates microcosms of the natural world to understand animal behavior.

Diving in with Nachum Ulanovsky
With an eye toward realism, the neuroscientist, who has a new study about bats out today, creates microcosms of the natural world to understand animal behavior.
Gene-activity map of developing brain reveals new clues about autism’s sex bias
Boys and girls may be vulnerable to different genetic changes, which could help explain why the condition is more common in boys despite linked variants appearing more often in girls.

Gene-activity map of developing brain reveals new clues about autism’s sex bias
Boys and girls may be vulnerable to different genetic changes, which could help explain why the condition is more common in boys despite linked variants appearing more often in girls.